Clinical Chemistry Test 4

A breakdown product of hemoglobin is:

Hemoglobin S can be separated from hemoglobin D by:

On electrophoresis at alkaline pH, which of the following is the slowest migrating hemoglobin?

The hemoglobin that is resistant to alkali (KOH) denaturation is:

The bilirubin results shown in the table are obtained on a patient:

day result

1. 4.3 mg/dL (73.5 μmol/L)
2. 4.6 mg/dL(78.7 μmol/L)
3. 4.5 mg/dL (77.0 μmol/L)
4. 2.2 mg/dL(37.6 μmol/L)
5. 4.4 mg/dL (75.2 μmol/L)
6. 4.5 mg/dL (77.0 μmol/L)

Given that the controls are within range each day, what is a probable explanation for the result on day 4?

Urobilinogen is formed in the:

In bilirubin determinations, the purpose of adding a concentrated caffeine solution or methyl alcohol is to:

If the total bilirubin is 3.1 mg/dL (53.0 μmol/L) and the conjugated bilirubin is 2.0 mg/dL (34.2 μmol/L), the unconjugated bilirubin is:

The principle of the tablet test or bilirubin in urine or feces is:

Ten heparinized plasma samples are assayed for bilirubin by Jendrassik-Grof method on analyzer 1,and by the Evelyn-Malloy method on analyzer 2, in a method comparison study.

The results are all 10-20% higher from analyzer 2 most likely because:

Serial bilirubin determinations performed by Jendrassik-Grof method are shown in this table:

The best explanation for the results is:

In the liver, bilirubin is converted to:

In which of the following disease states is conjugated bilirubin a major serum component?

Kernicterus is an abnormal accumulation of bilirubin in:

In which of the following conditions does decreased activity of glucuronyl transferase result in increased unconjugated bilirubin and kernicterus in neonates?

A 21-year-old man with nausea, vomiting, and jaundice has these laboratory findings:

These can best be explained as representing:

A stool specimen that appears black and tarlike should be tested for the presence of:

What substance gives feces its normal color?

A condition in which erythrocyte protoporphyrin is increased is:

The definitive diagnosis for hereditary coproporphyria (HCP) is a markedly increased: